On Adaptation of Adenosine Monophosphate-Activated Protein Kinase to Exercise Skeletal Muscle 腺苷酸基琥珀酸合成酶腺苷酸活化蛋白激酶与运动性骨骼肌适应
Adenosine monophosphate-activated protein kinase ( AMPK). 服用爱卡,目的是专杀一种名叫一磷酸腺苷活性蛋白酶的蛋白质(AMPK)。
RT-PCR was used to examine the gene transcripts concentrations of adenosine kinase ( AK) and hypoxanthine-guanine phosphoribosyltransferase ( HGPRT) in each group. 应用RT-PCR法检测各实验组腺苷激酶(AK)和次黄嘌呤-鸟嘌呤磷酸核糖转移酶(HGPRT)基因转录产物含量的变化。
Objectiv To investigate the role of cyclic adenosine monophosphate ( cAMP) and protein kinase C ( PKC) in the phagocytic process of human retinal pigment epithelial ( HRPE) cells by measuring phagocytosis indexes in specific and nonspecific phagocytic process. 目的研究人视网膜色素上皮(HRPE)细胞特异性及非特异性吞噬过程中环磷酸腺苷(cAMP)浓度及蛋白激酶C(PKC)活性的变化,探讨cAMP及PKC在HRPE的吞噬功能中所起的作用。
BACKGROUND& OBJECTIVE: Adenosine plays an essential role in cell s signal transduction, including inhibiting the activity of phosphatidylinositol 4-kinase, up-regulating the activity of adenylate cyclase and the concentration of cAMP, and inhibiting platelet actin polymerization. 背景与目的:腺苷作用于细胞信号转导系统,可抑制磷脂酰肌醇4-激酶活性、升高腺苷酸环化酶活性和细胞内cAMP水平、抑制血小板肌动蛋白聚合等。
Adenosine kinase ( AK), a key enzyme in the regulation of the cellular concentrations of adenosine ( A), is an important physiological effector of many cells and tissues. 腺苷激酶(adenosinekinase,AK)是控制细胞中腺苷浓度的一种关键酶,在许多细胞和组织中发挥着重要的生理效应子作用。
Adenosine monophosphate-activated protein kinase disease mimicks hypertrophic cardiomyopathy and Wolff-Parkinson-White syndrome: Natural history 腺苷酸活化蛋白激酶病酷似肥厚性心肌病和预激综合征:自然病史
The alteration of cyclic adenosine monophosphate and protein kinase C in the phagocytic process of human pigment epithelial cells 人视网膜色素上皮细胞吞噬过程中环磷酸腺苷及蛋白激酶C的改变
Preconditioning by Isoflurane Induces Lasting Sensitization of the Cardiac Sarcolemmal Adenosine Triphosphate-sensitive Potassium Channel by a Protein Kinase C-δ-mediated Mechanism 异氟醚预处理通过激活蛋白激酶C-δ诱发心肌纤维膜ATP敏感性钾通道持久敏感化
Genetics characteristics of erythrocyte acid phosphatase, adenosine deaminase and adenylate kinase in the Han population of west Guangdong province 广东粤西地区汉族人群红细胞酸性磷酸酶-腺苷脱氨酶-腺苷酸激酶的遗传学特性
Azathioprine and its potential metabolite inhibits adenosine kinase, diminish AMP formation by adenosine. 硫唑嘌呤及其有效代谢物抑制腺苷激酶(AK),使Ado转变AMP减少,从而增加Ado浓度。
By using the method of documentary, the paper summarizes the adaptation mechanism of Adenosine Monophosphate-Activated Protein Kinase ( AMPK) to exercise skeletal muscle. 通过文献检索方法对腺苷酸活化蛋白激酶参与运动引起骨骼肌适应的基因表达调节机制进行综述。
Adenosine receptors are coupled to G proteins, though altering the activity of a second messenger system, can modulate protein kinase signaling pathways and regulate downstream target gene expression. 腺苷受体与G蛋白偶联,通过改变第二信使系统活性来调节胞内蛋白激酶信号通路和调控下游靶基因表达。
However the CAMP ( cyclic adenosine monophosphate)-PKA ( protein kinase A) and PI3K ( phosphatidylinositol-3 kinase)-AKT signaling pathway played an important role in Rg1 induced NSCs proliferation. 而CAMP(环磷酸腺苷)-PKA(蛋白激酶A)和PI3K(磷脂酰肌醇-3激酶)-AKT信号传导通路在Rg1促增殖过程中发挥重要作用。